Last Chance! IMPC survey closing in August!

The IMPC is at a funding crossroads and seeking community input.  Help shape the future of the IMPC by taking part in our short survey.

Setmar | SET domain without mariner transposase fusion

GeneMGI:1921979Genome BrowserSynonyms: 5830404F24Rik, Etet2

Physiological systems

21 / 24 physiological systems tested

7 Significantly impacted by the knock-out

 Homeostasis/metabolism Pigmentation Integument Vision/eye Hematopoietic system Behavior/neurological Skeleton

14 No significant impact

3 Not tested

Data collections

Viability data
Body weight measurements
Embryo imaging data
Gene metrics:14Significant phenotypes
0Associated diseases
Expression examined in:45Adult tissues
0Embryo tissues

Phenotypes

  Loading...










* This parameter was manually assessed for significance.
Download data as:  

lacZ Expression

Associated images

Loading...

Human diseases caused by Setmar mutations

The analysis uses data from IMPC, along with published data on other mouse mutants, in comparison to human disease reports in OMIM, Orphanet, and DECIPHER.

Phenotype comparisons summarize the similarity of mouse phenotypes with human disease phenotypes.






Download data as:  

Histopathology

Loading...

IMPC related publications

Loading...

Order Mouse and ES Cells

Loading...

The IMPC Newsletter

Get highlights of the most important data releases, news and events, delivered straight to your email inbox

Subscribe to newsletter